2014, Volume 7, Issue 2, pp 192 – 201

Pneumonias – a dynamic relationship between the human organism and the etiologic agent

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Authors and Affiliations

Correspondence to:Radu Chiotan, MD, Primary doctor, Respiratory medicine, Assistant professor, Carol Davila University of Medicine and Pharmacy "Prof. Dr. Marius Nasta" Institute for Respiratory Medicine, 90 Viilor Av., District 5, Bucharest Phone: +40213362571, Mobile phone: +0744308903, E-mail: raduchio@yahoo.com

Abstract

Abstract

Pneumonias are usually acute infectious processes of the pulmonary parenchyma, being the result of the overcome of the natural defence mechanisms of the human organism, which lead to the penetration, persistence and multiplication of a biologic agent (which has become a pathogen agent) within the lungs. This is a phenomena which generates local manifestations (inflammation) and systemic manifestations, their variable dimension (which is conditioned by the state of the host and/or the virulence of the pathogen agent) explaining the clinical, paraclinical and biological dimensions of the phenomena. The current material deals on one hand with the phenomena which takes part in the formation of the heterogeneous spectrum of the pneumonias and, on the other hand, we will demonstrate the manner in which the expansion and the severity of the infectious phenomena depend on the virulence of the etiologic agent, on the manner in which the defence mechanisms have been overcome, and also on the biological state of the invaded organism.

Abbreviations: PN – pneumonia, O2 – oxygen, CO2 – carbon dioxide

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About this article

PMC ID: 4197495
PubMed ID: 25408724
DOI: 

Article Publishing Date (print): 15-06-2014
Available Online: 25-06-2014

Journal information

ISSN Printing: 1844-122X
ISSN Online: 1844-3117
Journal Title: Journal of Medicine and Life

Copyright License: Open Access

This article is distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use and redistribution provided that the original author and source are credited.


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